A younger organism would naturally release more cytokines than and elderly one.

年轻的有自然会比年老的有放更多的细胞因。

IL-5, on the other hand, activates eosinophils which promote an immune response by releasing more cytokines and leukotrienes.

另一方面，IL-5激活嗜酸性粒细胞，通过放更多的细胞因和白三烯来促进免疫反应。

Cytokines are thought to also enhances the activity of the NMDA receptors.

这些细胞因可能使NMDA受的活性增强。

The release of cytokines causes symptoms like fever, drenching night sweats, and weight loss.

细胞因的放会导致发烧、夜间盗汗和重减轻等症状。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T细胞会产生一些小的信号分，即细胞因，激活B细胞和巨噬细胞。

It works as an anti-inflammatory and similar to other remedies, influences cytokine production in the body.

它可以作为一种抗炎药，和其他药物一样，能够影响内细胞因的产生。

Cytokines are proteins released by cells to interact with each other and to trigger specific processes.

细胞因是细胞间相互作用并触发特定过程而放的蛋白质。

Known for its antibacterial activity, it increases the release of inflammatory cytokines, which signal cells to repair themselves.

众所周知，它具有抗菌活性，能增加炎症细胞因的放，而炎症细胞因是细胞自的信号。

Dr Robert Snelgrove from Imperial College London, among others, has theorised the notion of the so-called 'cytokine storm'.

伦敦帝国理工学院的罗伯特·斯内尔格罗夫博士等人提出了所谓“细胞因风暴”理论。

Along with an increase in proteins called cytokines—which the researchers say might suggest a boost in immune activity.

同时一种名为细胞因的蛋白质含量升高，对此研究人员表示这可能表明免疫活性提升。

In response, the injured cells release inflammatory molecules called cytokines that activate the immune system to repair the injury.

对此反应，损伤的细胞放出 称为“细胞因”的发炎分用来激活免疫系统和损伤。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

细胞因也会趋化B淋巴细胞和巨噬细胞，这也是炎症反应的一部分。

If the T cell gets activated, it kicks into action, producing cytokines which helps to get other immune cells involved.

T细胞在被激活后，开始合成分泌细胞因，进而调动其他更多的免疫细胞。

If it hijacks the immune system, and a cytokine storm occurs?

如果被它操控了整个免疫系统 并且发生了细胞因风暴？

In chronic disease states, chemical messengers called cytokines mediate this pathologic process in the kidney, immune system, and the GI tract.

在慢性疾病中，称为细胞因的化学信使在肾脏、免疫系统和胃肠道中介导了这一病理过程。

Afterwards, the macrophages release protein molecules called cytokines whose job is to recruit and organize more virus-busting cells from your immune system.

然后巨噬细胞会放一种叫作细胞因的蛋白质分它会从免疫系统中征集并组织更多的细胞来杀死病毒。

In some cases, the nucleus macrophages attack the nucleus pulposus, and start secreting inflammatory cytokines, like tumor necrosis factor alpha, an interleukin-1, and interleukin-6.

在某些情况下，巨噬细胞会攻击髓核，并分泌炎性因，比如肿瘤坏死因α（TNF-α）、白细胞介素-1（IL-1）和白细胞介素-6（IL-6）。

Another cytokine called IL-10 mediates the expression of increased ferritin receptors on the surface of macrophages, which then sequesters even more iron.

另一种叫做白介素-10的细胞因介导巨噬细胞表面的铁蛋白受的表达增加，如此一来，铁蛋白受会螯合更多的铁。

Alcohol also contributes to pancreatitis in other ways, though, for example, stimulating acinar cells to release inflammatory cytokines which attracts a strong immune reaction.

酒精还可以通过其它方式导致胰腺炎，比如促进腺泡细胞放炎性细胞因，从而引起强烈的免疫反应。

Soon enough, the handful of white blood cells surveilling the CSF identify the pathogen and release cytokines to recruit additional immune cells.

之后很快，一些负责监视脑脊液的白细胞识别出病原，并放细胞因召集额外的免疫细胞。