A younger organism would naturally release more cytokines than and elderly one.

年轻的有机体自然会比年老的有机体释放更多的因子。

IL-5, on the other hand, activates eosinophils which promote an immune response by releasing more cytokines and leukotrienes.

另一方面，IL-5激嗜酸性粒，通过释放更多的因子和白三烯来促进免疫反应。

The release of cytokines causes symptoms like fever, drenching night sweats, and weight loss.

因子的释放会导致发烧、夜间盗汗和体重减轻等症状。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T会产生一些小的信号分子，即因子，激B和巨。

Known for its antibacterial activity, it increases the release of inflammatory cytokines, which signal cells to repair themselves.

众所周知，它具有抗菌性，能增加症因子的释放，而症因子是自我修复的信号。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

因子也会趋化B淋巴和巨，这也是症反应的一部分。

Along with an increase in proteins called cytokines—which the researchers say might suggest a boost in immune activity.

同时一种名为因子的蛋白质含量升高，对此研究人员表示这可能表明免疫性提升。

In response, the injured cells release inflammatory molecules called cytokines that activate the immune system to repair the injury.

对此反应，损伤的释放出 称为“因子”的发分子用来激免疫系统和修复损伤。

If the T cell gets activated, it kicks into action, producing cytokines which helps to get other immune cells involved.

T在被激后，开始合成分泌因子，进而调动其他更多的免疫。

In chronic disease states, chemical messengers called cytokines mediate this pathologic process in the kidney, immune system, and the GI tract.

在慢性疾病中，称为因子的化学信使在肾脏、免疫系统和胃肠道中介导了这一病理过程。

In some cases, the nucleus macrophages attack the nucleus pulposus, and start secreting inflammatory cytokines, like tumor necrosis factor alpha, an interleukin-1, and interleukin-6.

在某些情况下，巨会攻击髓核，并分泌性因子，比如肿瘤坏死因子α（TNF-α）、白介素-1（IL-1）和白介素-6（IL-6）。

Afterwards, the macrophages release protein molecules called cytokines whose job is to recruit and organize more virus-busting cells from your immune system.

然后巨会释放一种叫作因子的蛋白质分子它会从免疫系统中征集并组织更多的来杀死病毒。

Alcohol also contributes to pancreatitis in other ways, though, for example, stimulating acinar cells to release inflammatory cytokines which attracts a strong immune reaction.

酒精还可以通过其它方式导致胰腺，比如促进腺泡释放性因子，从而引起强烈的免疫反应。

Soon enough, the handful of white blood cells surveilling the CSF identify the pathogen and release cytokines to recruit additional immune cells.

之后很快，一些负责监视脑脊液的白识别出病原体，并释放因子召集额外的免疫。

In response to hypoxia, myocardial cells release signalling molecules called cytokines, which cause dilation of the segment of coronary artery B beyond the blockage.

为了应对缺氧，心肌会释放因子这类信号分子，使冠脉B阻塞处远端的血管扩张。

Newer therapies called biologics can also be useful, because they block the actions of cytokines like tumor necrosis factor alpha and the interleukins.

生物制剂治疗这种新疗法同样有效，能阻断TNF-α和IL-1等因子的作用。

That's because cytokines and prostaglandin can reach even higher structures in your brain, disrupting the activity of neurotransmitters, like glutamate, endorphins, serotonin, and dopamine.

这是因为因子和前列腺素可以到达你大脑中更高的结构，破坏神经递质性，如谷氨酸，内啡肽，血清素，和多巴胺。

Second, cytokines travel through the body to the hypothalamus, the part of the brain responsible for controlling temperature, thirst, hunger, and sleep, among other things.

其次，因子穿过身体到达下丘脑，它是大脑中控制温度调节，口渴，饥饿，在其他事情中还有，睡意。

The cytokines attract Inflammatory cells like macrophages which start releasing even more inflammatory substances like proteases, platelet activating factor, and free radicals, all which contribute to inflammation.

这些因子可以募集更多的症如巨；这些巨进而释放更多的症介质，如蛋白酶、血小板化因子和自由基，它们都可以进一步促进症反应。

Since they are previously exposed, the immune system's memory t-cells quickly release cytokines to try and control the new outbreak which forms more areas of caseous necrosis.

因为宿主之前曾经接触过分枝杆菌，免疫系统中的记忆T快速释放因子来试图控制住新一轮疾病的暴发，而这个过程会导致更多的肺部组织形成干酪样坏死。