When pathogens enter the lungs, the macrophages work to destroy them.

当病因抵达肺时，巨噬细胞就开始摧毁它们。

These helper T-cells produce small signaling molecules called cytokines, which activate B-cells and macrophages.

辅助T细胞一些小的信号分子，即细胞因子，活B细胞和巨噬细胞。

In this stage the exudate gets digested by enzymes, ingested by macrophages, or coughed up.

在这个阶段，分泌物被酶分解，由巨噬细胞吞噬或咳出。

Cells called macrophages summon all sorts of others to an injury, to try to repair it.

巨噬细胞召集其他免疫细胞到达受伤处，并试图修复伤害。

They cytokines also attract B-cells and macrophages as part of the inflammatory reaction.

细胞因子也趋化B淋巴细胞和巨噬细胞，这也是炎症反应的一分。

As a result, Legionella is able to survive and replicate inside macrophages.

军团菌从而得以在巨噬细胞内存活并增殖。

First, specialized cells called macrophages eat the invading material in an attempt to clean up the inflammatory mess.

首先，巨噬细胞吃掉外来物质，试图清理有可能出现的炎症。

Some monocytes migrate into tissues and differentiate into macrophages, which remain in tissues and aren’t found in the blood.

一些单核细胞迁移到组织中，分化成为巨噬细胞；它们停留在组织里，不再在血液里出现。

The laser beam breaks the ink globules into smaller particles that can then be cleared away by the macrophages.

颜料块击碎，变成更小的颗粒，巨噬细胞可以将其清除。

But macrophage signalling also involves SNAP-25, so Dr Chapman thinks he can use botulinum toxin to shut the cells up.

但是巨噬细胞的信号通路中也包括SNAP-25，因此查普曼博士想，他能够用肉毒素来关闭这些免疫细胞。

Cells called macrophages charge in as the first line of attack, searching for and destroying the viruses and infected cells.

巨噬细胞作为第一线的攻击者，将搜索并消灭病毒与受感染的细胞。

Phagocytosis of apoptotic cells or bodies by adjacent healthy cells, either parenchymal cells or macrophages.

凋亡小体在溶酶体内迅速降解，相邻细胞通过迁移或增殖以取代现已清除的凋亡细胞占据的原有空隙。

Alveolar macrophages then eat up these red blood cells, which causes them to take on this brownish color from iron build-up.

肺泡巨噬细胞之后吞噬这些红细胞，导致这些细胞由于铁元素积聚而呈现棕褐色。

Afterwards, the macrophages release protein molecules called cytokines whose job is to recruit and organize more virus-busting cells from your immune system.

然后巨噬细胞释放一种叫作细胞因子的蛋白质分子它从免疫系统中征集并组织更多的细胞来杀死病毒。

That’s because there are a few “tingible bodies” which are macrophages that have eaten up dead neoplastic cells that look like little stars.

这是因为有一些着色小体巨噬细胞，也就是巨噬细胞，吞噬凋亡的肿瘤细胞后，而看似像小星星。

This means that it protects the bacteria against phagocytosis by macrophages and neutrophils, allowing Klebsiella to escape destruction.

它可以保护细菌免受巨噬细胞和中性粒细胞的吞噬，使克雷伯杆菌不能被消灭。

Another cytokine called IL-10 mediates the expression of increased ferritin receptors on the surface of macrophages, which then sequesters even more iron.

另一种叫做白介素-10的细胞因子介导巨噬细胞表面的铁蛋白受体的表达增加，如此一来，铁蛋白受体螯合更多的铁。

So with iron either being locked up in macrophages, or unable to be absorbed, fully productive hemoglobin laden RBCs cannot be produced.

铁要么被锁在巨噬细胞中，要么无法被吸收，因而无法满载血红蛋白的红细胞。

This channel serves as a pathway for iron to enter the macrophage at increased rates, so less iron is available for the production of hemoglobin.

这个通道使铁以更快的速度进入巨噬细胞，因此，可用于成血红蛋白的铁含量减少。

In some cases, the nucleus macrophages attack the nucleus pulposus, and start secreting inflammatory cytokines, like tumor necrosis factor alpha, an interleukin-1, and interleukin-6.

在某些情况下，巨噬细胞攻击髓核，并分泌炎性因子，比如肿瘤坏死因子α（TNF-α）、白细胞介素-1（IL-1）和白细胞介素-6（IL-6）。