1.Information about their glomerular target antigens is inconsistent, and whether aailability of target antigens, antibody specificity or aidity are nephritogenic parameters, is not determined.
1.关于肾小球靶抗原矛盾
,靶抗原
否有效、抗体特异
和力
否
致肾炎因素,这个也无法确定。
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18.When the abnormal IgA1-IgG immune complexes deposit in the mesangium, the alternative complement pathway is activated, leading to the release of proinflammatory cytokines and migration of macrophages into the kidney, all of which contributes to glomerular injury.
当异常的IgA1-IgG免疫复合物沉积在系膜上时,补体旁路途径被激活,导致促炎因子的释放,同时促进巨噬细胞向迁移,以上所有反应会导致
小球的损伤。