1.Another cause is Dressler syndrome which occurs several weeks after a myocardial infarction, or heart attack.

另个病因是发生于心肌梗死数周后心肌梗死后综合征。

2.And that can lead to a myocardial infarction, which is what we also call a heart attack.

这可能导致心肌梗塞，也就是我们所说心脏病发作。

3.An MI means myocardial infarction, just another name for a heart attack.

MI 表示心肌梗塞，只是心脏病发作别称。机翻

4.Okay, it's not an infarction. It's a dissection.

好吧 这是心肌梗塞 是夹层。

5.Basically, when heart cells die in a myocardial infarction, it leads to massive inflammation that also involves the serous pericardium.

心肌梗死导致心肌细胞死亡，引发累及膜心包强烈炎症反应。

6.In contrast partial occlusion may progress to localized infarction.

相反，部分闭塞可发展为局部梗死。

7.The susceptibility of the ventricular wall to this type of partial thickness infarction is not uniform.

心室壁对这种部分厚度梗死易感性致。

8.It should be noted, however, that this condition is clinically unstable and often precedes a myocardial infarction.

心肌肌钙蛋白水平保持正常，或只是轻微升高。然而，应该指出是，这种疾病在临床上是稳定，而且常常发生在心肌梗死之前。

9.In severe cases, blood flow can been completely cut off, which results in myocardial infarction, or the necrosis of a portion of myocardium.

严重情况下，血流会被完全阻断，引起心肌梗死，即心肌部分坏死。

10.Okay, let's recap - acute pericarditis is usually caused by a idiopathic causes, but can be caused by viruses or after a myocardial infarction.

急性心包炎通常原因未明，也可因病毒感染或心肌梗死导致。

11.Then entire thickness of the myocardial wall supplied by that vessel become ischemic, and is at risk of infarction.

然后，由该血管供应整个心肌壁就会缺血，具有梗死风险。

12.In the absence of treatment, the process may progress to complete transmural infarction of the area involved with development of pathological Q waves.

在没有治疗情况下，这过程可能进展到与病理性Q波发展有关区域完全跨壁梗死。

13.A prolonged ischemia can lead to death of the heart tissue, known as heart attack or myocardial infarction.

血液供应减少称为缺血。长时间缺血可导致心脏组织死亡，即心脏病发作或心肌梗死。

14.The ECG changes associated with infarction in this situation are identical to those of pure ischemia with depression of the ST segment, T-wave inversion or both.

这种情况下与梗死相关心电图变化与单纯缺血伴ST段压低、T波倒置或两者兼而有之心电图变化相同。

15.So we've seen that in ST elevation MI, untreated a complete arterial occlusion may progress to infarction of the full thickness of the affected myocardial wall.

所以我们已经看到，在ST段抬高型心肌梗死，未经治疗完全性动脉闭塞可能发展为心肌壁全厚度梗死。

16.STEMI is infarction that extends the entire thickness of the myocardium.

STEMI指心肌梗死延伸至心肌全层。

17.In inferior infarction, the injury current represented here by the blue arrows, therefore produces ST elevation in these leads.

下壁梗死时，受损电流用蓝色箭头表示，因此在这些导联产生ST段抬高。

18.The location of infarction can be identified based on the leads with ST elevation.

通常在电相反导联上有个相互ST段压低。

19.As shown here, as the process of infarction evolves the T waves may demonstrate a further abnormality developing ash a variable time point after occlusion.

如图所示，随着梗死过程发展，T波可能在堵塞后任时间点出现进步异常发展。

20.Most patients with infarction show elevated troponin levels within 6 hours.

大多数脑梗死患者在6小时内肌钙蛋白水平升高。