The lipotropic effect of threonine and lysine might result from their enhancement of fatty acid oxidation activityin liver mitochondria.
其机转可能细胞粒体中之氧化作用。
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In IgA nephropathy, though, there is abnormal glycosylation of these serine and threonine residues, causing them to be galactose-deficient.
IgA肾病中,有一系列丝酸、酸残基异常糖基,导致它们无法连接半乳糖。
Among these are serine and threonine residues which are O-linked glycosylated, meaning they have a sugar molecule, specifically galactose, attached to their oxygen.
其中有丝酸酸残基,是o链糖基的,这意味着它们的氧上有一个糖分子,半乳糖居多。
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